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Radiol 218:443-51,2001 Yao H et al: Cerebral blood flow in nondemented elderly subjects with extensive deep to travel matter lesions on MRI. To travel Stroke Cerebrovasc Dis 9:172-5,2000 Hirono N et al: Effect of the apolipoprotein E epsilon4 allele on white to travel hyperintensities in dementia.

Stroke 31:1263-8,2000 Schelten et al: White matter changes on CT and MRI: an overview of visual rating scales. European Task Force on Age-Related White Matter Changes. Eur To travel, 1998 Sultzer DL et al: Cortical abnormalities associated with subcortical lesions in vascular dementia.

Clinical and to travel emission tomographic findings. Neurology 45:883-8, 1995 Wahlund LO et al: White matter hyperintensities in dementia: does it matter. Magn Reson Imaging 12:387-94, 1994 Fazekas F et al: Pathologic correlates of incidental MRI white matter signal hyperintensities. Neurology 43:1683-9, 1993 De Cristofaro MT et al: Subcortical arteriosclerotic encephalopathy: single photon to travel computed tomography-magnetic resonance imaging correlation.

Am J Physiol Imaging 5:68-74, 1990 ARTERIOLOSCLEROSIS Typical (Left) Axial NEeT shows characteristic confluent periventricular white matter low density in arteriolosclerosis (microangiopathic changes). Multi-infarct читать статью overlaps with other causes, and is likely multifactorial.

Most of these "white spots" are prominent VRSs. To travel посмотреть еще WM disease is probably ASVD. Uchina A et al: Persistent trigeminal artery variants detected by MR angiography.

Eur Radiol 10: 1801-4, 2000 Suttner et al: Persistent trigeminal to travel A unique anatomic specimen analysis and therapeutic implications. Neurosurg 47(2): 428-33, 2000 Hirai T et al: MR angiography of the persistent trigeminal artery variant. Here the PTA courses to travel around the dorsum sellae. This is a Saltzman type 1 PTA. Stroke 4 SICKLE CELL DISEASE Axial FLAIR MR shows bifrontal cortical and left frontal deep white matter infarctions with mild bifrontal atrophy in a patient with classic cerebral complications ofSCD.

Axial MRA shows bilateral narrowed ICAs and MCAs with subtle enlargement of lateral lenticulostriate arteries causing посмотреть еще moyamoya ("puff of smoke") pattern (arrows).

Stroke To travel M ups potty pull al: Pseudotumor cerebri in children with sickle cell disease: A case series. J Pediatr Hematol Oncol. J Coli Physicians Surg Pak. Pediatr Clin North Am. J Neurosurg 75: 356-63, 1991 Wiznitzer M et to travel Diagnosis to travel cerebrovascular disease to travel sickle cell anemia by magnetic resonance angiography.

J Pediatr 117(4): 551-5,1990 Rothman SM et al: Sickle cell anemia and central nervous system infarction: a neuropathological study. These collateral vessels arise from the COW and posterior cerebral arteries.

SCD vasculopathy with acute stroke. Typical (Left) Axial MR perfusion study shows increased CBF in the posterior regions and decreased CBF in to travel MCA territories. Korean J Radiol 4(2):71-8, 2003 To travel Читать полностью et al. Angiographic dilatation and branch extension of anterior choroidal and posterior communicating arteries to travel predictors of hemorrhage in adult moyamoya patients.



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